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p-jak1 primary antibody  (Keygen Biotech)

 
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    Keygen Biotech p-jak1 primary antibody
    P Jak1 Primary Antibody, supplied by Keygen Biotech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/p-jak1 primary antibody/product/Keygen Biotech
    Average 90 stars, based on 1 article reviews
    p-jak1 primary antibody - by Bioz Stars, 2026-03
    90/100 stars

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    primary antibodies against p-janus kinase 1 (jak1)  (Santa Cruz Biotechnology)
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    MicroRNA-199a modulates the progression of CRC cells via the <t>PI3K/AKT</t> and JAK/STAT signaling pathways. A, The miR-199a overexpression reduces PI3K/AKT signaling through ROCK1 downregulation. B, Overexpression of miR-199a suppresses JAK/STAT signaling through ROCK1 downregulation. C, Exogenetic overexpression of ROCK1 partially restores the miR-199a-induced inactivation of PI3K/AKT and JAK/signal transducing activator of transcription signaling pathways. CRC indicates colorectal cancer; JAK1, Janus kinase 1; miR-199a, microRNA-199a; PI3K, phosphoinositide 3-kinase; ROCK1, Rho-associated coiled coil-containing protein kinase 1; STAT, signal transducing activator of transcription.
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    Image Search Results


    Western blotting analysis of signalling proteins related to the JAK1/STAT6 and TLR signalling pathway in M2 microphages. ( A ) The levels of the signalling proteins JAK1/p-JAK1, STAT6/p-STAT6 and β-catenin in M0 macrophages and M2 macrophages; ( B ) the levels of the signalling proteins JAK1/p-JAK1, STAT6/p-STAT6 and β-catenin in M2 macrophages after α-MMC treatment; ( C ) the levels of the signalling proteins AP1/p-AP-1, TAK1/p-TAK1, p65/p-p65 in M2 macrophages after α-MMC treatment; ( D – F ) Relative density of p-JAK1/ JAK1, p-STAT6/ STAT6 and β-catenin/β-actin in M0 macrophages and M2 macrophages; ( G – I ) Relative density of p-JAK1/ JAK1, p-STAT6/ STAT6 and β-catenin/β-actin in M2 macrophages after α-MMC treatment; ( J – L ) relative density of p-AP1/AP-1, p-TAK1/TAK1, p-p65/p65 in M2 macrophages after α-MMC treatment. The data shown are individual values with the mean ± SEM; n = 3. *P < 0.05 significantly different from the control group; n.s. no significant difference from the control group. One-way analysis of variance, Tukey’s multiple comparison tests.

    Journal: Journal of Inflammation Research

    Article Title: Alpha-Momorcharin Inhibits Proinflammatory Cytokine Expression by M1 Macrophages but Not Anti-Inflammatory Cytokine Expression by M2 Macrophages

    doi: 10.2147/JIR.S372306

    Figure Lengend Snippet: Western blotting analysis of signalling proteins related to the JAK1/STAT6 and TLR signalling pathway in M2 microphages. ( A ) The levels of the signalling proteins JAK1/p-JAK1, STAT6/p-STAT6 and β-catenin in M0 macrophages and M2 macrophages; ( B ) the levels of the signalling proteins JAK1/p-JAK1, STAT6/p-STAT6 and β-catenin in M2 macrophages after α-MMC treatment; ( C ) the levels of the signalling proteins AP1/p-AP-1, TAK1/p-TAK1, p65/p-p65 in M2 macrophages after α-MMC treatment; ( D – F ) Relative density of p-JAK1/ JAK1, p-STAT6/ STAT6 and β-catenin/β-actin in M0 macrophages and M2 macrophages; ( G – I ) Relative density of p-JAK1/ JAK1, p-STAT6/ STAT6 and β-catenin/β-actin in M2 macrophages after α-MMC treatment; ( J – L ) relative density of p-AP1/AP-1, p-TAK1/TAK1, p-p65/p65 in M2 macrophages after α-MMC treatment. The data shown are individual values with the mean ± SEM; n = 3. *P < 0.05 significantly different from the control group; n.s. no significant difference from the control group. One-way analysis of variance, Tukey’s multiple comparison tests.

    Article Snippet: The recommended dilution ratios of the JAK1/p-JAK1, STAT6/p-STAT6 and β-catenin primary antibodies are JAK1 (Immunoway, YT4535, 1:1000), P-JAK1 (Immunoway, YP0424, 1:1000), STAT6 (Abcam, ab32520, 1:1000), P-STAT6 (Abcam, ab263947, 1:1000), and β-catenin (Proteintech, 51067-2-AP, 1:4000).

    Techniques: Western Blot

    MicroRNA-199a modulates the progression of CRC cells via the PI3K/AKT and JAK/STAT signaling pathways. A, The miR-199a overexpression reduces PI3K/AKT signaling through ROCK1 downregulation. B, Overexpression of miR-199a suppresses JAK/STAT signaling through ROCK1 downregulation. C, Exogenetic overexpression of ROCK1 partially restores the miR-199a-induced inactivation of PI3K/AKT and JAK/signal transducing activator of transcription signaling pathways. CRC indicates colorectal cancer; JAK1, Janus kinase 1; miR-199a, microRNA-199a; PI3K, phosphoinositide 3-kinase; ROCK1, Rho-associated coiled coil-containing protein kinase 1; STAT, signal transducing activator of transcription.

    Journal: Technology in Cancer Research & Treatment

    Article Title: MiR-199a-5p Inhibits the Growth and Metastasis of Colorectal Cancer Cells by Targeting ROCK1

    doi: 10.1177/1533034618775509

    Figure Lengend Snippet: MicroRNA-199a modulates the progression of CRC cells via the PI3K/AKT and JAK/STAT signaling pathways. A, The miR-199a overexpression reduces PI3K/AKT signaling through ROCK1 downregulation. B, Overexpression of miR-199a suppresses JAK/STAT signaling through ROCK1 downregulation. C, Exogenetic overexpression of ROCK1 partially restores the miR-199a-induced inactivation of PI3K/AKT and JAK/signal transducing activator of transcription signaling pathways. CRC indicates colorectal cancer; JAK1, Janus kinase 1; miR-199a, microRNA-199a; PI3K, phosphoinositide 3-kinase; ROCK1, Rho-associated coiled coil-containing protein kinase 1; STAT, signal transducing activator of transcription.

    Article Snippet: After blocking with 5% nonfat milk, the membranes were incubated with primary antibodies against ROCK1, p-PI3K (Tyr458), PI3K, p-AKT (Ser473), AKT, p-Janus kinase 1 (JAK1), JAK1, p-signal transducing activator of transcription (STAT) 3, STAT3, and GAPDH (1:1000; Santa Cruz Biotechnology, Santa Cruz, California) at 4°C overnight.

    Techniques: Protein-Protein interactions, Over Expression